Ground-breaking discoveries by United States researchers
could eventually help protect New Zealand honeybees from the
deadly varroa mite that has killed off many of the nation's
feral bees and damaged the economics of running apiaries.
Michigan State University researchers have been able to
produce in the laboratory for the first time proteins that
help channel sodium ions through cell membranes of varroa
mites - which could lead to development of a specific varroa
control which would not affect bees, according to the
research, which was published in the Journal of Biological
Chemistry.
In New Zealand most varroa control is done with chemical
miticides such as fluvalinate, which paralyses the mite and
eventually kills it.
However, there are growing concerns that varroa may develop
resistance to fluvalinate (sold as Apistan) - something which
is already happening overseas, including in the United
States.
"The insecticide used to control varroa mites, fluvalinate,
targets the mite sodium channel," said Professor Ke Dong, an
entomologist.
"Successfully producing the mite sodium channel in the lab
now allows scientists to develop new chemicals that target
the mite sodium channel but don't affect the honeybees."
The researchers also found an amino acid in the mite sodium
channel that made the pest resistant to tetrodotoxin, or TTX,
a deadly poison found in puffer fish not currently used as an
insecticide.
TTX is thought to be the toxin in grey side-gilled sea slugs
which has been killing dogs on Auckland and Coromandel
beaches - it is apparently present in beach sediments.
Varroa mites can kill an entire hive within a year, feeding
on bee blood and transmitting viruses.
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